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ekceli qiqi pn xsure whether allergy or ape..tp sejak sebulan lalu kaki (dr ujung kaki sampai pangkal paha) jd gatal2..mcm ruam jugak,ade consult doc tp dia cume bg lotion ape tah yg kaler pink 2..pakai mcm bedak air..unfortunately smpai skg still xsembuh,n my legs look very bad..huhu...mlm nk tdo pn terganggu sbb terlalu gatal,bile pagi mandi mesti pedih je..huhu  |
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assalamualaikum,
setahu kaklia la kalu salah tlg btlkan allergi ni dapat dikenalpasti melalui ujian2 makmal yg dilakukan. Antaranya patch test,photo patch test.prick test dan ujian total igE.
So daripada test ni kita boleh tahu kulit kita allergik kepada apa bahan.Setahu kaklia test2 ni kat swasta mahaiii tapi kat kerajaan murah jek.Cuma bila kita dah tahu apa bahan yang kita allergi tu kita kena hindarkanlah bahan itu.rasanya itu saja caranya.
Patch test ni kita boleh wat gak kat kosmetik yg kita guna,ubatan dan mcm2 la yg kita rasa badan kita allergik pada ?.harap korang paham la seposen ye keterangan seposen akak ni..... |
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Reply 36# iman_kasih
setau aku autoimmune ni bahay..mmg ada allergic yg autoimmune klu tak silap tp bkn semua...
rheumatoid arthritis, lupus tu antara penyakit yg disebabkan oleh autoimmune
yup, betul |
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Reply 25# anuar
iman...,simptom allergic ni autoimune..nak increase apa lagi...?sepatutnya distabilkan imune system
Anuar, just curious, you boleh tolong quotekan tak which reference or textbook yang you refer to come up with this statement? So that i can open exactly the same book yang u refer... |
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saya baca buku tajuk atlas imunology second edition..
Penulis buku tu Robert E,Lewis dan Julius M.Cruse..-Professor Of Pathology.Dept Of Pathology,Director Of Immunopathology and transplanttation,
Professor of medicine and prof of microbiology...Univ of Missisippi.. |
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Reply 36# iman_kasih
sikit sikit autoimune...iman...ko orang sendiri yg cerdik pandai dlm medical pun menjawap persoalan ttg allergic ni mcm tak ada keyakinan....tak percaya statement I ni?...cuba teliti balik semua...kalo allergic tu autoimune kata kan dengan yakin yes...kalo bukan say no!
Iman,I nak U buat pencerahan...I tak faham maksud imune respon dlm kes allergic ni...
IMAN...,APE SAL LAK AWAK PETIK TF..?apa benda nya TF tu?(U jawap kat pm. je...) |
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ko fahamkan balik je apa maksud autoimmne..kan ko jgk yg kata allergy ni autoimmune..tu yg aku jwb BUKAN SEMUA allergy ni disebabkan oleh autoimmune mechanism. auto immune tu satu mechanism dlm bhs mudahnya mcm mech yg salah faham ttg sel/tisu/organ sendiri menyebabkan ia menyerang sel/tisu/organ tersebut..
ko ada ilmu apa salahnya ko share kat sini...kalau kedekut tak payah cakap byk beb..
pasal tf sbb ak pnh jumpa ko promote tf dekat thread lain.. |
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source: http://www.aarda.org/q_and_a.php
What is autoimmunity?
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One of the functions of the immune system is to protect the body by responding to invading microorganisms, such as viruses or bacteria, by producing antibodies or sensitized lymphocytes (types of white blood cells). Under normal conditions, an immune response cannot be triggered against the cells of one's own body. In certain cases, however, immune cells make a mistake and attack the very cells that they are meant to protect. This can lead to a variety of autoimmune diseases. They encompass a broad category of related diseases in which the person's immune system attacks his or her own tissue.
What causes autoimmunity?
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The immune system normally can distinguish "self" from "non-self." Some lymphocytes are capable of reacting against self, resulting in an autoimmune reaction. Ordinarily these lymphocytes are suppressed. Autoimmunity occurs naturally in everyone to some degree; and in most people, it does not result in diseases. Autoimmune diseases occur when there is some interruption of the usual control process, allowing lymphocytes to avoid suppression, or when there is an alteration in some body tissue so that it is no longer recognized as "self" and is thus attacked. The exact mechanisms causing these changes are not completely understood; but bacteria, viruses, toxins, and some drugs may play a role in triggering an autoimmune process in someone who already has a genetic (inherited) predisposition to develop such a disorder. It is theorized that the inflammation initiated by these agents, toxic or infectious, somehow provokes in the body a "sensitization" (autoimmune reaction) in the involved tissues.
What are the types of autoimmunity?
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Particular autoimmune disorders are frequently classified into organ-specific disorders and non-organ-specific types. Autoimmune processes can have various results, for example, slow destruction of a specific type of cells or tissue, stimulation of an organ into excessive growth, or interference in its function. Organs and tissues frequently affected include the endocrine gland, such as thyroid, pancreas, and adrenal glands; components of the blood, such as red blood cells; and the connective tissues, skin, muscles, and joints. Some autoimmune diseases fall between the two types. Patients may experience several organ-specific diseases at the same time. There is, however little overlap between the two ends of the spectrum.
In organ-specific disorders, the autoimmune process is directed mostly against one organ. Examples, with the organ affected, include Hashimoto's thyroiditis (thyroid gland), pernicious anemia (stomach), Addison's disease (adrenal glands), and type 1 diabetes (pancreas).
In non-organ-specific disorders, autoimmune activity is widely spread throughout the body. Examples include rheumatoid arthritis, systemic lupus erythematosus (SLE or lupus), and dermatomyositis.
What are some of the treatments for autoimmune diseases?
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Of first importance in treating any autoimmune disease is the correction of any major deficiencies. An example would be replacing hormones that are not being produced by the gland, such as thyroxin in autoimmune thyroid disease or insulin in type 1 diabetes. In autoimmune blood disorders, treatment may involve replacing components of the blood by transfusion.
Second in importance is the diminishing of the activity of the immune system. This necessitates a delicate balance, controlling the disorder while maintaining the body's ability to fight disease in general. The drugs most commonly used are corticosteroid drugs. More severe disorders can be treated with other more powerful immunosuppressant drugs, such as methotrexate, cyclophosphamide, and azathioprine. All of these drugs, however, can damage rapidly dividing tissues, such as the bone marrow, and so are used with caution. Intravenous immunoglobulin therapy is used in the treatment of various autoimmune diseases to reduce circulating immune complexes. Some mild forms of rheumatic autoimmune diseases are treated by relieving the symptoms with nonsteroidal anti-inflammatory medications. Drugs that act more specifically on the immune system, for example, by blocking a particular hypersensitivity reaction, are being researched.
What is the family connection in autoimmune diseases?
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The ability to develop an autoimmune disease is determined by a dominant genetic trait that is very common (20 percent of the population) that may present in families as different autoimmune diseases within the same family. The genetic predisposition alone does not cause the development of autoimmune diseases. It seems that other factors need to be present as well in order to initiate the disease process. It is important for families with members who have an autoimmune disease to mention this fact when another member of the family is experiencing medical problems that appear to be difficult to diagnose. |
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List of Autoimmune and Autoimmune-Related Diseases
Acute Disseminated Encephalomyelitis (ADEM)
Acute necrotizing hemorrhagic leukoencephalitis
Addison's disease
Agammaglobulinemia
Allergic asthma
Allergic rhinitis
Alopecia areata
Amyloidosis
Ankylosing spondylitis
Anti-GBM/Anti-TBM nephritis
Antiphospholipid syndrome (APS)
Autoimmune aplastic anemia
Autoimmune dysautonomia
Autoimmune hepatitis
Autoimmune hyperlipidemia
Autoimmune immunodeficiency
Autoimmune inner ear disease (AIED)
Autoimmune myocarditis
Autoimmune pancreatitis
Autoimmune retinopathy
Autoimmune thrombocytopenic purpura (ATP)
Autoimmune thyroid disease
Axonal & neuronal neuropathies
Balo disease
Behcet’s disease
Bullous pemphigoid
Cardiomyopathy
Castleman disease
Celiac disease
Chagas disease
Chronic fatigue syndrome**
Chronic inflammatory demyelinating polyneuropathy (CIDP)
Chronic recurrent multifocal ostomyelitis (CRMO)
Churg-Strauss syndrome
Cicatricial pemphigoid/benign mucosal pemphigoid
Crohn’s disease
Cogans syndrome
Cold agglutinin disease
Congenital heart block
Coxsackie myocarditis
CREST disease
Essential mixed cryoglobulinemia
Demyelinating neuropathies
Dermatitis herpetiformis
Dermatomyositis
Devic's disease (neuromyelitis optica)
Discoid lupus
Dressler’s syndrome
Endometriosis
Eosinophilic fasciitis
Erythema nodosum
Experimental allergic encephalomyelitis
Evans syndrome
Fibromyalgia**
Fibrosing alveolitis
Giant cell arteritis (temporal arteritis)
Glomerulonephritis
Goodpasture’s syndrome
Graves' disease
Guillain-Barre syndrome
Hashimoto's encephalitis
Hashimoto’s thyroiditis
Hemolytic anemia
Henoch-Schonlein purpura
Herpes gestationis
Hypogammaglobulinemia
Idiopathic thrombocytopenic purpura (ITP)
IgA nephropathy
IgG4-related sclerosing disease
Immunoregulatory lipoproteins
Inclusion body myositis
Insulin-dependent diabetes (type1)
Interstitial cystitis
Juvenile arthritis
Juvenile diabetes
Kawasaki syndrome
Lambert-Eaton syndrome
Leukocytoclastic vasculitis
Lichen planus
Lichen sclerosus
Ligneous conjunctivitis
Linear IgA disease (LAD)
Lupus (SLE)
Lyme disease, chronic
Meniere’s disease
Microscopic polyangiitis
Mixed connective tissue disease (MCTD)
Mooren’s ulcer
Mucha-Habermann disease
Multiple sclerosis
Myasthenia gravis
Myositis
Narcolepsy
Neuromyelitis optica (Devic's)
Neutropenia
Ocular cicatricial pemphigoid
Optic neuritis
Palindromic rheumatism
PANDAS (Pediatric Autoimmune Neuropsychiatric Disorders Associated with Streptococcus)
Paraneoplastic cerebellar degeneration
Paroxysmal nocturnal hemoglobinuria (PNH)
Parry Romberg syndrome
Parsonnage-Turner syndrome
Pars planitis (peripheral uveitis)
Pemphigus
Peripheral neuropathy
Perivenous encephalomyelitis
Pernicious anemia
POEMS syndrome
Polyarteritis nodosa
Type I, II, & III autoimmune polyglandular syndromes
Polymyalgia rheumatica
Polymyositis
Postmyocardial infarction syndrome
Postpericardiotomy syndrome
Progesterone dermatitis
Primary biliary cirrhosis
Primary sclerosing cholangitis
Psoriasis
Psoriatic arthritis
Idiopathic pulmonary fibrosis
Pyoderma gangrenosum
Pure red cell aplasia
Raynauds phenomenon
Reflex sympathetic dystrophy
Reiter’s syndrome
Relapsing polychondritis
Restless legs syndrome
Retroperitoneal fibrosis
Rheumatic fever
Rheumatoid arthritis
Sarcoidosis
Schmidt syndrome
Scleritis
Scleroderma
Sjogren's syndrome
Sperm & testicular autoimmunity
Stiff person syndrome
Subacute bacterial endocarditis (SBE)
Susac's syndrome
Sympathetic ophthalmia
Takayasu’s arteritis
Temporal arteritis/Giant cell arteritis
Thrombocytopenic purpura (TTP)
Tolosa-Hunt syndrome
Transverse myelitis
Ulcerative colitis
Undifferentiated connective tissue disease (UCTD)
Uveitis
Vasculitis
Vesiculobullous dermatosis
Vitiligo
Wegener’s granulomatosis
**NOTE Fibromyalgia and Chronic Fatigue are listed, not because they are autoimmune, but because many persons who suffer from them have associated autoimmune disease(s)
American Autoimmune Related Diseases Association
22100 Gratiot Avenue, Eastpointe, MI 48021-2227
www.aarda.org |
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setakat ni tu yg dpt aku tempek ye so enjoy la membaca ye... |
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Reply 48# iman_kasih
iman,dimana titik tolak persamaan isu autoimune ni?
Mmg saya ada promote Tf kat thread sebelah..adakah I dah lakukan si thread ni?
Iman nak kan perkongsian ilmu tapi sikap awak tak mencerminkan mahukan perkongsian itu...
Ada promoter Sne promote dgn link sekali....awak dah sound? kalo i promote Tf disini pasti awak dah pangkah!so dimana pertimbangan keterbukaaan perkongsian ilmu? |
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Reply 50# iman_kasih
sekian punya panjang list penyakit..so tak mustahil physician terkeliru ttg penyakit dan simptom...persoalannya..Apakah ubat ape paling sesuai dan ape peranan ubat ubat tu?
Mintak pencerahan plssss... |
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Reply 45# cn7
buku atlas imunology third edition kata gastrik adalah autoimune jugak.... |
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Reply 54# anuar
Which page... in our hospital we dont use that kind of reference. Unless lab based. |
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Reply 46# anuar
Penulis buku tu Robert E,Lewis dan Julius M.Cruse..-Professor Of Pathology.Dept Of Pathology,Director Of Immunopathology and transplanttation,
Professor of medicine and prof of microbiology...Univ of Missisippi..
dude,
boleh scan & tempek kat sini?
and, 1 buku jer ker? Consensus expert panel lain takdak? |
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Reply 48# iman_kasih
ko ada ilmu apa salahnya ko share kat sini...kalau kedekut tak payah cakap byk beb..
pasal tf sbb ak pnh jumpa ko promote tf dekat thread lain..
tf ni amende? Promote tf? produk ker? |
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Reply 55# cn7
lab test pun ada gak....kalo harap buku tu je biAS... |
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gehhh...ggeehhh...ggeehhh..mild autoimune je lee...saya ada baca dlm paper konon nya penyakit ni "seribu wajah" pasal ia menyerang dlm pelbagai jenis penyakit.susah utk dikesan.
anuar,
actually what are you saying? i was referring to your statement pada iman kasih stating that allergic is autoimmune (or as you mentioned, mild autoimmune) because setahu i patophysiology of allergic is not due to autoimmune, but it is inter related. Now you're talking about gastrik & autoimmune jugak, so what is your point? |
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ab test pun ada gak....kalo harap buku tu je biAS.
that is why i ask if it is lab based ke tidak, because if it is, then it will be a whole lot other story.
contoh, in my hospital, when we do ward rounds, and patients perlukan antibiotik (abx) for a certain indicationor diagnosis, the ward round will involve the primer team, infectious disease specialist, pharmacist and microbiologist. Microbiologist most of the time will just refer on C&S culture that they have from the lab, but other professionals will look into clinical symptoms as well. meaning, the lab result might say drug A does not work for this patient, but if patient's clinical symptoms eg white cell count, temperature, ESR, CRP shows improvement, wound gets better etc, they will stick to the same drug & dont change it although the lab result (or microbiologists) says otherwise.
as you sendiri mention, to refer to 1 book is bias, because to come with a statement, it should be a collective consensus among the expert panels.
anyway, thank you for the info on the book. As you mentioned in your PM, the book is RM1000 (you kata murahkan). Of course, murahlah tu sebab medical text books are wayyyyyy more expensive.
i have consulted a few of my colleagues, but i have yet to consult my Professor in immunology yet (she's busy, banyak attend meetings & what not), but so far none of them can relate that allergic is caused by autoimmune.
personally, and this is just my opinion, (everyone is entitiled to their opinion, so you can backlash me afterwards), but i honestly dont think the way you ask iman kasih was because you really wanna know or share the knowledge, but its more of because you want to show that you can out-smart her...and this is just my opinion, your niat (of proving youmight be right but your WAY of doing it, a big question mark.
if you are a professional in medical background, then maybe we can talk the same language, not necessarily the same wavelength, but if this thread is gonna be used to show how brilliant someone else's is and others are not, then i'm walking away...
okay dude, the floor is yours ! |
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Post time 8-2-2011 01:35 PM
